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Antiques Roadshow guest stunned by HUGE value of jewellery with royal connection
By Tom Kucher
October 18, 2021
EntertainmentUSA
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AN ANTIQUES Roadshow guest was left stunned after the expert revealed the jaw-dropping value of some precious jewellery with a royal connection.
Expert Geoffrey Munn was joined by two ladies who brought along brooches had been handed down through their family.
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Expert Geoffrey Munn was joined by two guests with their broochesCredit: BBC
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The said their great-great-aunt was gifted the jewellery by Queen Alexandra of DenmarkCredit: BBC
The women explained that they items originally belonged to a great-great-aunt who worked for Queen Alexandra of Denmark.
He was thrilled to examine the jewellery, he said: “Well two jewels here, two original boxes for them, and in the lid, there are suppliers to the Royal Family.
“This one says Hancock’s and that one says Collingwood. Both of them have been suppliers to the British Royal Family for quite a long time.
“What amazing relics they are!”
The expert then took a closer look at each brooch and noted that one was made of pure gold and embellished with coral.
He explained that coral is associated with Venus, the goddess of love and could be an item you’d give to a potential suitor.
Geoffrey continued: “This is the sort of thing that would mark a big occasion in a courtier’s life.”
Moving onto the second brooch, he said: “This one is in black Onyx with a very conspicuous pearl cross on it.
“It ticks every single box for memorial jewellery. So, this is commemorating somebody that had died and I think if we turn it over we’ll be able to see the date in which that happened.”
He then turned over the brooch and uncovered a picture of Queen Alexandra, with delicate inscription.
It read: “From Her Royal Highness the Princess of Wales, July 7, 1870.”
An amazed Geoffrey said: “This is an intensely personal gift.”
The ladies added that their great-great-aunt was maid for the then Princess Alexandra and was gifted the jewellery.
The ladies nervously waited for the estimation, with Geoffrey telling them that the piece had significant history behind it.
Geoffrey said: “When you can find a piece of jewellery like this, with the maker, with the provenance, and the exact history, comes a jolly nice value.
“I have no hesitation at all in saying this is £6,000 to £8,000.”
The owners were blown away by the valuation, with one exclaiming: “Wow! That is amazing!
“I’m going to say £8,000 to £10,000.”
The huge sum initially left the guests lost for words, one replied: “That is incredible! Wow, that is amazing. I mean we are really – it’s the historical value of them.”
Geoffrey pointed out just how importance of these inscriptions, otherwise they would have been worth “a 10th of the value”.


Scientists need to conduct more research to understand how COVID-19 is linked to these neurological symptoms.
Researchers have presented their findings on these links at Neuroscience 2021, the annual conference of the Society for Neuroscience.
Researchers have been exploring the way COVID-19 affects a person’s nervous system, causing neurological symptoms.
This week, they presented their findings at Neuroscience 2021. This event is the annual meeting of the Society for Neuroscience, which is the largest global society for scientists and clinicians focused on issues of brain and nervous system health.
The findings indicate how SARS-CoV-2 — the virus that causes COVID-19 — gains access to a person’s brain. They also provide insight into the effects it has once it has entered this part of the body.
Neurological symptoms
For most people, the primary symptoms of COVID-19 are respiratory. According to the World Health Organization (WHO), the most common symptoms of COVID-19 are fever, blank pills in birth control a cough, tiredness, and the loss of taste or smell.
These symptoms and other, less common ones can range in severity from mild to life threatening. Some people who acquire a SARS-CoV-2 infection may even remain asymptomatic.
As well as these respiratory symptoms, clinicians and researchers have also observed various neurological symptoms associated with COVID-19.
Researchers have linked COVID-19 to headaches, brain inflammation, muscle weakness or pain, and numbness or weakness in a person’s hands, feet, or limbs.
However, further research is necessary to understand how COVID-19 affects a person’s nervous system.
Access to the brain
At Neuroscience 2021, a number of papers detailed findings exploring how SARS-CoV-2 is likely to affect a person’s brain.
Dr. Ashutosh Kumar, an assistant professor in the Department of Anatomy at the All India Institute of Medical Sciences, Patna, India, presented findings that identified a possible receptor that may give SARS-CoV-2 access to a person’s brain.
In other parts of the body, SARS-CoV-2 gains access to cells by binding to the protein angiotensin converting enzyme 2 (ACE2) with the help of transmembrane serine protease 2 (TMPRSS2). However, ACE2 and TMPRSS2 are not significantly present in a person’s brain.
Speaking with Medical News Today, Dr. Kumar said: “We know that COVID-19 patients show prominent neuropsychiatric symptoms and [that] virus-induced injuries — including evidence of the replicating virus in brain tissue — have been noted in the postmortem studies. However, a negligible expression of the ACE2 and TMPRSS2 left a key question [un]answered: How does SARS-CoV-2 enter the brain cells?”
“By [the middle] of the last year, another receptor — neuropilin-1 precursor (NRP1), a cell surface protein implicated in the brain development and causation of cancers — was shown capable of binding to furin-cleaved SARS-CoV-2 spike protein[s]. Uniquely, binding to the NRP1 didn’t require TMPRSS2.”
“By curiosity, we checked the expressions of NRP1 and furin in the postmortem human brain tissue, which [revealed] that both of these molecules are abundant in all key brain regions — chiefly, the olfactory and hippocampus.”
“Olfactory nerves present a way for the entry of the virus into the brain through the nose, and their infection can also explain the loss of smell as a key symptom in COVID-19. Further, the involvement of the hippocampus can explain the prominence of psychiatric symptoms in COVID-19 patients during and after recovery from acute illness,” said Dr. Kumar.
For Dr. Kumar, his team’s findings may help identify future drugs to protect against or treat the neurological symptoms of COVID-19.
“Our findings are important in the sense that they indicate the putative drug targets for preventing and treating neuropsychiatric symptoms in COVID-19. The binding sites of NRP1 and furin at the SARS-CoV-2 spike protein can be targeted with various approaches, such as with monoclonal antibodies or decoy receptors,” said Dr. Kumar.
The findings appear in both the Journal of Neuroscience Research and Frontiers in Immunology.
Monkey models
Prof. John H. Morrison, director of the California National Primate Research Center at the University of California, Davis, presented research on a study using rhesus monkeys. These findings also suggest that the olfactory system could be the way in which SARS-CoV-2 gains access to the brain.
Prof. Morrison told MNT: “The monkeys were infected both through the nose and the trachea and sacrificed [7] days after infection. We were particularly interested in whether or not aged type 2 diabetic (T2D) monkeys were more vulnerable to neuroinvasion by the virus than young monkeys, given that aging and T2D are key comorbidities in humans.”
“We studied the brains of four groups: 1) young [with neither the infection nor diabetes], 2) young [with the infection but without diabetes], 3) aged [with neither the infection nor diabetes], and 4) aged [with both the infection and diabetes]. The major findings are that:
For Prof. Morrison, the findings make it clear that COVID-19 is a neurological condition as well as a respiratory one.
“There has been an ongoing debate over whether or not SARS-CoV-2 can infect neurons,” said Prof. Morrison. “These findings demonstrate unequivocally that neurons can be infected in our monkey model of COVID-19 and that the virus appears to enter the brain through olfactory connections from the nose.”
“The direct entry through [the] olfactory system, productive infection of neurons, and transport to multiple brain regions by SARS-CoV-2 is the likely cause of neurological complications in COVID-19, such as anxiety, memory loss, and ‘brain fog.’ The heightened vulnerability of aged T2D monkeys suggests that such comorbidities in humans may also lead to more extensive neurological complications.”
“Thus, COVID-19 is potentially a brain disorder as well as a respiratory disease, and prevention and treatment of patients with long-term symptoms of COVID-19 should target protecting the brain as well as [the] heart and lungs. In addition, such treatments need to be applied very early after infection given that this occurred in [7] days.”
For Prof. Morrison, better understanding the neurological effects of COVID-19 is likely to help in researchers’ understanding of long COVID.
“Many of the symptoms associated with ‘long COVID’ are neurologic in nature and likely to be caused by neuroinvasion and infection of neurons. We do not know if such symptoms are linked to ongoing infection of neurons linked through circuits or through damage that occurred early and outlasts the infection,” said Prof. Morrison.
Prof. Morrison noted that the finding that older, T2D monkeys had more severe neurological infections was backed up by research in humans.
“Clinical studies have shown that [older people] are far more likely to display neurological symptoms. In addition, the virus is getting to some brain regions that are highly vulnerable to Alzheimer’s disease as well, but we don’t know if such an infection leaves a patient more vulnerable to Alzheimer’s disease in the future,” said Prof. Morrison.
Peripheral nervous system
In another study, researchers found that SARS-CoV-2 is also likely to be able to gain access to a person’s brain via nerves that convey touch and pain.
Jonathan Joyce — a graduate student in the laboratory of Dr. Andrea S. Bertke in the Department of Population Health Sciences at Virginia Tech, Blacksburg, VA — used a mouse model to explore whether SARS-CoV-2 could infect the peripheral nerves. Dr. Bertke worked closely with Joyce throughout the study.
Speaking with MNT, Joyce said that “[w]hile most of the attention paid to assessing the ability of SARS-CoV-2 to invade the nervous system has focused on the central nervous system — for example, the brain — we have found that SARS-CoV-2 can infect neurons of the peripheral nervous system.”
“Previous studies have shown that SARS-CoV-2 can infect nerves in the peripheral nervous system, but they only looked at nerves responsible for smell.”
“We have shown that SARS-CoV-2 is capable of infecting additional nerves of the peripheral nervous system in the head, neck, and body responsible for carrying out automatic bodily — for example, superior cervical ganglion — functions and relaying sensory information, particularly those that convey touch and pain, to the brain and spinal cord — for example, trigeminal ganglion and dorsal root ganglion.”
“We also found that various regions of the brain, impacted in COVID-19, are capable of being infected by SARS-CoV-2. It is worth noting that these findings are based on a mouse model of infection and will have to be repeated in other animal models for verification,” said Joyce.
According to Joyce, the value of the research is that it demonstrates that SARS-CoV-2 is not restricted to infecting the central nervous system but can also infect the peripheral nervous system at multiple points.
“Many studies that investigate how SARS-CoV-2 enters the nervous systems focus on how it enters the brain using neurons in the nose responsible for smell,” said Joyce.
“Our findings show that the virus can infect the peripheral nervous system at multiple points, particularly among nerves that convey touch and pain, and that some of these points have connections to regions of the brain impacted by COVID-19.”
“Presence of the virus in these nerves may contribute to the symptoms [that] people with COVID-19 [experience].”
For live updates on the latest developments regarding the novel coronavirus and COVID-19, click here.


Barriers and Cultural Influences on Sport and Exercise participation Medical Conditions: Medical conditions are a barrier in sport and exercise participation as they usually reduce someone’s ability to compete in a sport at either to a required level or even completely leave them unable to participate in a sport. Medical conditions could apply to people with either a physical or mental disability which includes for example people with amputated limbs which is a physical disability and also cerebral palsy which is a mental disability.
Medical conditions stop participation in sport in many ways, whether an individual can compete at a certain level of a sport or any form of the sport at all really depends on the type and severity of their medical condition. An example of a sportsperson experiencing a barrier through their medical condition would be Oscar Pistorius who is a Paralympic sprinter and has lost both lower parts of his legs. The barrier which he experiences is that he cannot compete on the same level playing field as Olympic sprinters due to his condition.
There are 2 initiatives to overcome this barrier; the first being the creation of the Paralympics which allows disabled athletes to compete against other disabled athletes which allows for them to compete on a level playing field and also create more fair competition for the athletes. The other initiative is for prosthetic limbs to be created for Oscar Pistorius and other disable athletes which replicate actual limbs.
This creation of the prosthetic limbs allows people such as Oscar Pistorius to be able to compete alongside Olympic athletes should they be able to actually qualify and compete, even though there remains to be controversy surrounding this idea. I think that it is unfair for Oscar Pistorius to be allowed into Olympic competition as he will not experience fatigue in his lower legs like the other athletes will do, this will give him an advantage over the other athletes. Oscar Pistorius showing off his prosthetic limbs during one of his races
Fitness: Someone’s fitness or rather lack of fitness usually acts as a barrier to them in the sport of exercise they participate in. Someone’s lack of fitness can act as a barrier in their respective sport as it may not allow them to participate to their highest level or even a required level to play the sport or do exercise. A few examples of fitness problems which act as barriers to playing sport or doing exercise would be leg injuries for footballers, hand injuries for boxers and for most sports in general a lack of aerobic fitness.
A real life example of someone’s fitness acting as a barrier to them in their sport would be Abou Diaby who is a professional football which experiences a lot of injuries and fails to keep his fitness at the required level in which to play professional football. A way in which this problem can be tackled would be the physios at his football club having a wide knowledge of how to treat his problems and then be able to maintain his fitness once the player reaches a level of fitness required to play professional football.
The picture below shows all of the injuries which Diaby has suffered throughout his career and highlights his difficulty in getting fit to play football. http://epltalk. com/2013/04/03/all-of-abou-diabys-35-injuries-for-arsenal-from-2006-2013-infographic/ Resources: The lack of resources can act as a barrier in sports participation as some sports require necessary equipment in order to play the sport e. g. o play football you must have at least a football or to play tennis you at least require to rackets and a ball. If you do not have the necessary equipment then you cannot possibly compete in the sport. These barriers could apply to lots of people around the world; mainly people of poor backgrounds who cannot afford the resources, another group who may be affected by lack of resources would be lower level sports clubs who do not have the money to acquire and maintain their resources.
An initiative which is put in place to aid lower level football clubs is the availably of funds by the FA (Football Association) available to lower level football facilities which the facilities can apply for and receive financial help from the FA in order to improve their facilities more and promote football activity in that facility, especially for the younger generations. http://www. thefa. com/my-football/football-volunteers/runningaclub/yourfacilities/funding Ethnic Origin: Ethnic origin is a clear cultural influence on sports participation.
The influences which this chosen cultural influence has on sports would be the amount of issues there are in sport surrounding racial hatred and discrimination. This group of people tends to participate in most sports these days but the sport black people for example would tend to participate in would be sports which require explosive speed and power and also height. Sports which require these things may include sprinting events such as 100m, basketball and boxing as these sports require a balance of speed, power and height.
The reason black people participate in these sorts of sports is because they are genetically superior to most other humans with a different ethnic background in general at the specific sports. This may influence decisions in sports participation as other people of different ethnic backgrounds may become intimidated by the large amount of black people taking part in whatever sport they are participating in. People may not think they have much chance in their sport if they come up against a large amount of black people who are known for being superior at whatever sport they are participating in.
An example of this cultural influence would be the clear lack of white people who participate in basketball. This lack of white people partaking in basketball activities is a mixture of them not just being physically inferior when it comes to the sport of basketball but they are also I think intimidated by the large number of black people and small number of white people in the NBA, this leaves them doubting their abilities and chances of making it in basketball which makes them not want to play them sport.
The table below shows the stats of African compared to Caucasian people who take part in some NBA teams, there is a clear difference in the number of African people compared to Caucasians. Team| African| Team| Caucasian| Portland Trailblazers| 93. 8| Milwaukee Bucks| 38. 3| LA Clippers| 92. 5| Minnesota Timberwolves| 36. 2| Boston Celtics| 91. 9| Orlando Magic| 33. 7| Washington Wizards| 91. 1| Phoenix Suns| 30. 7| Miami Heat| 90. 6| Houston Rockets| 30. 2| Detroit Pistons| 89. 7| New Orleans Hornets| 27. 1| Philadelphia 76ers| 88. 8| Chicago Bulls| 25. 2| Oklahoma City Thunder| 87. 9| LA Lakers| 24. 7|
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Cambiar título "Emby" y ícono "favicon.ico" Emby 4.4.3 - APORTE
Alexmanafan posted a topic in Spanish
Hola a todos, les traigo un aporte y espero sea permitido por la comunidad Emby. Se trata de cambiar el favicon.ico, pero principalmente de cambiar el título "Emby", esto debido a que hay muchos usuarios reportando o preguntando como se hace esto, y la verdad hay varias soluciones en la comunidad, pero practicamente a todas les falta siempre algún detalle. Por este motivo les traigo la solución definitiva y la que estoy utilizando actualmente. Lo más sencillo primero, Cambiar favicon.ico Lógicamente lo primero es tener nuestro nuevo ícono a mano. (extensión .ico) Bien copiamos nuestro nuevo favicon.ico, y nos vamos al directorio /system/dashboard-ui/ allí damos clic derecho Pegar nos pedirá confirmación para reescribir, damos Sí. Cambiar el título "Emby" por el nombre de nuestro servicio o servidor ejemplo "Server-multimedia" Lo primero que tienes que tener en cuenta es que necesitas editar 3 archivos y dentro de cada uno de ellos, debes buscar la palabra Emby varias veces, yo te indido... Nota: Realizar una salva de cada archivo que edites por si te equivocas, volver al original. 1er archivo a editar: /system/dashboard-ui/index.html dentro de este archivo index.html debemos buscar la línea: <meta name="application-name" content="Emby"> y reemplazarla por: (Server-Multimedia es el nombre que quieres poner) <meta name="application-name" content="Server-Multimedia"> Buscamos la línea: <meta property="og:title" content="Emby"> y la reemplazamos por: <meta property="og:title" content="Server-Multimedia"> Buscamos la línea: <meta property="og:site_name" content="Emby"> y la reemplazamos por: <meta property="og:site_name" content="Server-Multimedia"> Por último buscamos la línea: <title>Emby</title> y la reemplazamos por: <title>Server-Multimedia</title> Guardamos los cambios. 2do archivo a editar: /system/dashboard-ui/manifest.json Buscamos la línea: "name": "Emby", y la reemplazamos por: "name": "Server-Multimedia", Buscamos la línea: "short_name": "Emby", y la reemplazamos por: "short_name": "Server-Multimedia", Guardamos los cambios. 3er archivo a editar: /system/dashboard-ui/modules/appheader.js Buscamos el parámetro: document.title="Emby"}, y lo reemplazamos por: document.title="Server-Multimedia"}, Hay que tener cuidado con este archivo appheader.js debido a que no es por líneas sino que parece todo un párrafo, por eso debemos buscar bien el texto citado. Guardamos los cambios. En la mayoría de los casos debes, limpiar los datos del sitio en el navegador que usas, y reiniciar el servidor Emby. Al actualizar de versión, debemos realizar nuevamente los pasos, y es posible que cambien las ubicaciones o nombres de los archivos editados. (ya ha sucedido) Saludos y espero les resulte útil. Probado en Emby server 4.4.3.
August 14, 2020
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HI everyone,About character renaming refresh metadata
Joechung posted a topic in QNAP
Regarding the character's name being changed manually and refreshing the metadata, the name still cannot be restored. How can I restore the character's name to be the same as the original TMDB data? I tried manual identification and metadata refreshing and overwriting many times, but I couldn't restore to the original state of TMDB. Is there a way to restore the original name? Because I modified a lot of personal names and wanted to restore all the changed character names back to the original state of TMDB, I wanted to find a way to restore the character names. Looking forward to your reply. thanks
November 5
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Completed Add 'Sort by Filename' to the Browser
topbanana posted a topic in Feature Requests
When browsing movies, etc, add to the "AZ" Sorting pull-down menu, an additional option to "Sort by Filename" This then orders all the movies in the raw, default way, such that they match the file system on which the actual media files are stored, on disk. This order is generally quite different to the default sort order, as movie titles are sorted ignoring the articles: A, An, The... and also some movies have different names displayed, especially foreign films. The file systems sort by the basic pure alphanumeric order.
April 1, 2017
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TS-873: reading embedded <title> for nfo failed emby 4.3.1
quake posted a topic in QNAP
Hi, I am using QNap TS-873 since 2 weeks. I have installed emby 4.3.1 successfully. Now, I have moved my files (home videos) of the windows PC to the NAS. Unfortunately every displayed title of the mp4 files contains the filename. Imagine: I am recording my game session playin' Quake2. After gaming I tag / enrich the mp4 files with title, subtitle, description, marks in the file's detail. Using the windows version, the title will correct displayed in Emby, using the same in QNap Emby, the filename is displayed not the title or description. How can I correct or solve this? Thx
December 20, 2019
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Add many name too a same movie
khenghis posted a topic in General/Windows
Hi, Im not sure i'm posting in the right section off the forum its actually the first time I do so. I was just wonderring if by any chance there was a way to add multiple title to a same movie. In simple, I have some movie in a bylingual version and for some of them I reffer too their title in french and some other in english. Would it be possible to add 2 tittle to a movie in order that when i'm searching for it in my searchbar, event if its sorted name is his original one in english I may still find it with his french name.
August 17, 2019
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[4.1.0.26] Latest version doesn't expose the title of what's playing anymore?
RedaLazri posted a topic in MacOS
Hello, I'm running Emby server on a Qnap and accessing it via Chrome on macOS, I use it to listen to stuff as well as watching all sort of videos. One thing I have set up is Timing, a time tracker to see how much time I spend watching TV, working...etc. Before 4.1.0.26 Timing was able to catch the name of playing video and turn them to keywords to track them, since the update I only Get Emby 1h 30m which isn't good since I do many things while Emby is open not just watch TV. What's happened? Before After Note that I'm NOT running any Beta version, it's from another software and happens to have a close duration.
April 25, 2019
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Need help with adding movie year and resolution to the movie title
slickspidey posted a topic in DLNA
Hi, I'm pretty new to Emby, just got it today, and I'm wondering if there's a way to add a movie's year and resolution in the title? Just because I have multiple movie files that I watch on my TV through the DLNA media server, and I sometimes have multiple copies of the same movie because I sometimes get the 4k, 3D, and a normal 1080p copy. So ideally I'd like to display "Doctor Strange (2016) [4k]" "Doctor Strange (2016) [3D] "Doctor Strange (2016) [1080p]" as the title that way I can see clearly which file I want to play instead of having to play them all and guess which one is which. Then also the year of the movie would be beneficial for me when I have my collections to see at a glance how old or new movies are. Is there a way to do this with Emby? I don't want to change the original movie name because they're torrents and I'm seeding them and the torrent client would detect it as missing files and stop seeding once it does. I like it the way Emby currently has it where I see the cleaned up name on my TV through my DLNA media server and at the same time it doesn't interfere with the original movie name on my PC so I can keep seeding without interruption. I figure Emby already extracted the metadata for the movie year and resolution because they display it, so there must be a way to display movie year and resolution in the movie title when I view it on my TV. Help with this would be amazing!
January 31, 2019
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Duplicate Cut Title Sequence out of all TV episodes
ragmanii posted a topic in Feature Requests
I would like to cut the opening sequence from my tv shows. Usually this is just the same title sequence used either by the entire series or season. Here is how I see it envisioned: cut the video into three parts. Beginning, title then end. The beginning and the end would be specified by another sequence number from: Firefly - S01E14 - The Message Bluray-1080p.mp4 to: Firefly - S01E14 SEQ1 - The Message Bluray-1080p.mp4 and Firefly - S01E14 SEQ2 - The Message Bluray-1080p.mp4 Those to will contain the main episode the first can be the before the title and the second after the title. Then create one file that is like: Firefly - S01 Title - The Message Bluray-1080p.mp4 that contains just the title sequence. Doing it this way would allow us to cut out a lot of overlap in video's thus saving space and allowing a new option from the interface to not play the Title at all and just watch the "meat" of the show if desired. If the title is at the beginning of the show then SEQ1 wouldn't be needed and only the SEQ2 file would be there. This could also be expanded to cover the credits, but those usually change as guest stars could be listed, but it could be another option that uses the same technique. It may not seem like much but cutting a one minute title out of 22 episodes would save 21 minutes of hard drive. With the amount of content I have that is significant value.
December 21, 2018
1 reply
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Improve reporting features
topbanana posted a topic in General/Windows
Your libraries of movies, TV, etc. almost certainly have lots and lots and lots of misidentified items, errors... 5%, perhaps more is possible... And 5% of a large collection, is a lot of items. Wrongly identified. Emby relies on the various databases on the internet, and between them, they inevitably make the odd mistake... That's fair enough. Many factors are involved, bad filenames, rare titles, foreign character, etc, etc... Some are just stupidly wrong. They're going to happen... BUT. We still don't have any tools to find them. You have no way of knowing how many errors are in your libraries. We have to rely on stumbling upon the error, or if you have an intimate knowledge of what titles you do have, you'll spot a few more of the errors perhaps. As attempted a couple of times, some of us have tried combining the grossly, needlessly inadequate 'Reports' feature (now a plugin), with data pulled from the database files with external tools. This has worked OK, but is a royal PITA, and not slick, not quick to do. But it does show you how many errors emby holds. Which might horrify/surprise you. If you want some fun, reinstall emby from scratch, rebuilding your libraries... You'll very quickly see some errors, with many more being there without you knowing. ALL WE WANT is the ability to, at least, list the path, filename, against the title and year from metadata downloaded... This simple feature, ability, would make finding these numerous errors possible, if a little bit slow to process (takes a lot of concentration to check 5,000 filenames, titles, years). Later, yeah, it would be sweet to have it highlight non-exact matches, that would speed up the process immensely. But we need the minimum now. These tools need to be in the core of emby... It's not good enough that this media database application doesn't have the most basic data analysis tool! Path, filename, title, year. Please.
October 5, 2018
25 replies
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errors
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Add Sort by Filename/Path
topbanana posted a topic in Feature Requests
Now that the SortRemoveWords hack doesn't work, can we please, please just have Sort by 'Filename/Path' added to the sorting options! It's such a simple and obvious feature. I need to change the sort order to match the media files on the filesystem. But Emby sorts the titles ignoring the articles, A, An, The... Which is entirely valid, but we need to switch between either. Emby was created to help us wrangle huge numbers of media files, sorting data is such a basic feature! Please!
August 10, 2018
5 replies
1
sort order
sorting
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Metadata - Making "Original Title" a little more useful
anderbytes posted a topic in Feature Requests
I'd like to suggest some improvements for the "Original Title" field in Emby. - Make it searchable - Make it possible for Emby admin to choose which one ("Title" or "Original Title") appears in UI (default: "Title") - If possible, make it automatically fetch title metadata of the country where the item was produced, if the provider sends it.
May 31, 2016
5 replies
title
metadata
Can I Batch Edit Titles?
JXL2112 posted a topic in General/Windows
First: So I went ahead and edited all my album.nfo's adding a symbol for 16-bit music....of course Emby just wiped them over. I really don't want to edit "ALL" the titles one by one inside Emby...that would take forever! Is there a way to batch edit all of them or maybe just tag them (16-bit)?  Second: What symbol will display properly in the Roku app, It seems the symbol I chose doesn't display right? This is mostly a Roku App issue and not being able to see what type of album I'm playing.
April 19, 2018
12 replies
Edit Titles
Title
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Roku
Bug - Browser title bar not always updated
anderbytes posted a topic in General/Windows
This probably is simple, and has to be done. There are lots of screens in Emby that DON'T update the browser title bar. That leaves some outdated info in title bar when we go browsing Emby. In general... what I've noticed: - Screens of Server Management updates the title bar - Movie/Episode screen updates the title bar - Several screens of "Emby Home" panel doesn't updates the title bar - Choosing a library doesn't updates the title bar - Changing tabs in Home doesn't updates the title bar - Choosing Metadata Manager doesn't updates the title bar - Choosing Reports doesn't updates the title bar
May 31, 2016
6 replies
3
browser
title
Identifying the misidentified (reports?)
topbanana posted a topic in Feature Requests
Going thru my movies and tv shows, i do sometimes find one that's been misidentified. Often it's just the year that's wrong, as the release date is often different from the year of production (stated in the end credits) which means that it gets identified as another movie by emby. Can we have a way of finding these/spotting these easier? Idea: use Reports? I'd like to be able to compare a list of the identified movie titles with a list of the filenames. So just adding a 'filename' column (perhaps a 'path' column as well) to the reports page will allow me to scan down the list and spot the wrongly identified movies much, much quicker. As a bonus feature, perhaps we could have a filter where if the title matches the filename exactly, then they wouldn't show up leaving only the films with subtly different titles, foreign titles, etc, plus the misidentified films we seek.
February 17, 2016
18 replies
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title
filename
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Music title construction issue
RedStripe posted a topic in General/Windows
I have several albums .flac encoded. Two of the albums have the following symptoms: 1) The album name is prepended with another version of the album name. 2) Each track is prepended with another version of the track name. 3) The album has an associated .cue and .m3u file 4) The prepended album name is the same as the TITLE in the .cue file 5) The prepended track name is the same as the corresponding TRACK TITLE in the .cue file I removed the .cue and .m3u files but the displayed values remain. Can someone please explain this to me? Where are the displayed values being derived from? Is there something I can do to resolve this display issue? Here are the associated .cue and .m3u files: "You aren't permitted to upload this kind of file" Here are some screen shots:
April 16, 2014
10 replies
flac
cue
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MB Server incorrectly parses titles for shows based on dates that also have 720p in the title
laserchicken posted a topic in General/Windows
Hi, I couldn't find this issue when searching so I hope it's not a duplicate. Scenario: I have some shows that make more sense to organize by date than by season/episode numbers. My formatting rules also append the quality to the end of the show title. For example: Some Daily Show - (2014-09-14) - Show Title Here - 720p Expected: MB Server determines what episode this is using the date Observed: MB Server uses the 720p at the end to determine that this is episode 720 of the show and therefore shows the wrong information (usually none), places it in the wrong order (it's usually all jumbled and at the top) and is generally confusing. I've been using MB since MB2 and shows processed by that have metadata from that time frame show up correctly, it just seems that MB3 is parsing the titles incorrectly. Is this a known issue? Is there either [a] some way to fix this or have MB Server ignore the 720p at the end? I remember that MB2 would ignore text in square brackets [], is there a way to add values to this list of text to omit (assuming MB3 does the same thing)? Does it do this omission before parsing the titles for episode information? Assuming this is still a feature, it might be helpful to have this functionality surfaced in the Server configuration. Thanks!
September 14, 2014
8 replies
parse
show
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title
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Multiple Views Of Same Collection
ElLoboSolitario posted a topic in General/Windows
With the old version of Media Browser I was able to configure it to show multiple views of the same folders (All Movies by Title, All Movies by Year, All Movies by Genre, All Movies by Date added, All Blu-ray Titles) With Media Browser Classic, if I attempt to add the same folders under a different collection name, I get an error stating that those folders have already been used. Furthermore, under the old version of Media Browser, I could see all my movies listed in one place. This new version lists them according to the folders where they are located (i.e., my movies are stored as: E:\1080p, E:\720p, G:\Video\Movies\SD 480p, G:\Video\Movies\HD\720p and G:\Video\Movies\HD\1080p.) This new version isn't very user friendly from what I've discovered as to the way it presents media collections. Strangely enough, my television series are under two different folders, yet they all display as one collection.


Agenesis of the corpus callosum (ACC) is a rare birth defect in which there is a complete or partial absence of the corpus callosum. It occurs when the development of the corpus callosum, the band of white matter connecting the two hemispheres in the brain, in the embryo is disrupted. The result of this is that the fibers that would otherwise form the corpus callosum are instead longitudinally oriented along the ipsilateral ventricular wall and form structures called Probst bundles.
Agenesis of the corpus callosum
Specialty
Neurology
In addition to agenesis, other degrees of callosal defects exist, including hypoplasia (underdevelopment or thinness), hypogenesis (partial agenesis) or dysgenesis (malformation).[1]
ACC is found in many syndromes and can often present alongside hypoplasia of the cerebellar vermis. When this is the case, there can also be an enlarged fourth ventricle or hydrocephalus; this is called Dandy–Walker malformation.[2]
Contents
1 Signs and symptoms
1.1 Associated brain anomalies
1.2 Associated syndromes and conditions
2 Causes
2.1 Ciliopathies: rare genetic disorders
2.2 Cocaine and other street drugs
3 Diagnosis
4 Treatment
5 Prognosis
6 Culture
7 Notes
8 External links
Signs and symptomsEdit
MRI images of three patients in the sagittal plane. A and B: Complete agenesis of the corpus callosum. C: Complete agenesis of both the corpus callosum and the anterior commissure.
Signs and symptoms of ACC and other callosal disorders vary greatly among individuals. However, some characteristics common in individuals with callosal disorders include vision impairments, low muscle tone (hypotonia), poor motor coordination, delays in motor milestones such as sitting and walking, delayed toilet training and dysautonomic symptoms such as low perception of pain or chewing and swallowing difficulties.[citation needed]
Laboratory research has demonstrated that individuals with ACC have difficulty transferring more complex information from one hemisphere to the other.[3] They also have been shown to have some cognitive disabilities (difficulty in complex problem solving) and social difficulties (missing subtle social cues), even when their intelligence quotient is normal.[3] Recent research suggests that specific social difficulties may be a result of impaired face processing.[4] The unusual social behavior in childhood often resembles that of an autism spectrum disorder.[citation needed]
Other characteristics sometimes associated with callosal disorders include seizures, spasticity, early feeding difficulties and/or gastric reflux, hearing impairments, abnormal head and facial features, and intellectual disability.[1]
Associated brain anomaliesEdit
Brain anomalies that can sometimes occur in syndromes that cause callosal disorders include:[1][5]
Aplasia of the cerebellar vermis
Chiari malformation
Colpocephaly
Dandy–Walker syndrome
Holoprosencephaly
Hydrocephalus
Neuronal migration disorders such as grey matter heterotopia
Schizencephaly
Associated syndromes and conditionsEdit
Some syndromes that frequently include ACC are:[citation needed]
Acrocallosal syndrome
Aicardi syndrome
Andermann syndrome
Donnai–Barrow syndrome
Dwarfism
FG syndrome
L1CAM syndrome
Microcephalic osteodysplastic primordial dwarfism type II
Mowat–Wilson syndrome
Oculocerebrocutaneous syndrome
Saal Bulas syndrome
Septo-optic dysplasia (optic nerve hypoplasia)
Shapiro syndrome
Vici syndrome
Some conditions that can sometimes be associated with ACC or other callosal disorders include:[citation needed]
1p36 deletion syndrome
13q deletion syndrome
CDK13-related disorder
Craniofacial abnormalities and other oral and maxillofacial pathologies
Fetal alcohol syndrome
Fetal warfarin syndrome
Genitopatellar syndrome
Gomez-Lopez-Hernandez syndrome
Joubert syndrome
Lujan–Fryns syndrome
Marden–Walker syndrome
Maternal nutritional deficiencies or infections
Metabolic disorders
Okamoto syndrome
Opitz G/BBB syndrome
Pascual-Castroviejo syndrome
Pitt–Hopkins syndrome
Sensenbrenner syndrome
Strømme syndrome
Triploid syndrome
Trisomy 9
Xia-Gibbs syndrome
CausesEdit
Agenesis of the corpus callosum is caused by disruption to development of the fetal brain between the 3rd and 12th weeks of pregnancy.[6] In most cases, it is not possible to know what caused an individual to have ACC or another callosal disorder. However, research suggests that some possible causes may include chromosome errors, inherited genetic factors, prenatal infections or injuries, prenatal toxic exposures, structural blockage by cysts or other brain abnormalities and metabolic disorders.[1]
Ciliopathies: rare genetic disordersEdit
Until recently, the medical literature did not indicate a connection among many genetic disorders, both genetic syndromes and genetic diseases, that are now being found to be related. As a result of new genetic research, some of these are, in fact, highly related in their root cause despite the widely varying symptoms apparent on clinical examination. Agenesis of the corpus callosum is one such disease, part of an emerging class of diseases called ciliopathies. The underlying cause may be a dysfunctional molecular mechanism in the primary cilia structures of the cell organelles that are present in many cellular types throughout the human body. The cilia defects adversely affect "numerous critical developmental signaling pathways" essential to cellular development and thus offer a plausible hypothesis for the often multi-symptom nature of a large set of syndromes and diseases. Known ciliopathies include primary ciliary dyskinesia, Bardet–Biedl syndrome, polycystic kidney and liver disease, nephronophthisis, Alström syndrome, Meckel–Gruber syndrome and some forms of retinal degeneration.[7]
Cocaine and other street drugsEdit
In utero exposure to cocaine, heroin, amphetamines and phenylpropanolamine can lead to agenesis of corpus callosum.[8]
DiagnosisEdit
Callosal disorders can be diagnosed through brain imaging studies or during autopsy.[6] They may be diagnosed through an MRI, CT scan, Sonography, prenatal ultrasound, or prenatal MRI.[1]
TreatmentEdit
There are currently no specific medical treatments for callosal disorders, but individuals with ACC and other callosal disorders may benefit from a range of developmental therapies, educational support, and services. It is important to consult with a variety of medical, health, educational, and social work professionals. Such professionals include neurologists, neuropsychologists, occupational therapists, physical therapists, speech and language pathologists, pediatricians, recreation therapists, music therapists, geneticists, social workers, special educators, early childhood intervention specialists, and caregivers for adults.[1]
PrognosisEdit
Prognosis varies depending on the type of callosal abnormality and associated conditions or syndromes. It is not possible for the corpus callosum to regenerate.[6] Neuropsychological testing reveals subtle differences in higher cortical function compared to individuals of the same age and education without ACC, although some individuals with callosal disorders have average intelligence and live normal lives.[citation needed]
CultureEdit
Kim Peek, inspiration for the film Rain Man and renowned for his savant abilities, was born with agenesis of the corpus callosum, along with macrocephaly and damage to the cerebellum.[9]
NotesEdit
^ a b c d e f "ACC - What is agenesis of the corpus callosum?". University of Maine. Retrieved April 11, 2010.
^ Aldinger, Kimberly A.; Doherty, Dan (October 2016). "The genetics of cerebellar malformations". Seminars in Fetal & Neonatal Medicine. 21 (5): 321–332. doi:10.1016/j.siny.2016.04.008. ISSN 1744-165X. PMC 5035570. PMID 27160001.
^ a b "Agenesis of the Corpus Callosum". Travis Research Institute. Archived from the original on July 17, 2010. Retrieved April 11, 2010.
^ "Corpus Callosum Research Program". California Institute of Technology. Archived from the original on 10 April 2010. Retrieved April 11, 2010.
^ Edwards, Timothy J.; Sherr, Elliott H.; Barkovich, A. James; Richards, Linda J. (2014-06-01). "Clinical, genetic and imaging findings identify new causes for corpus callosum development syndromes". Brain. 137 (6): 1579–1613. doi:10.1093/brain/awt358. ISSN 0006-8950. PMC 4032094. PMID 24477430.
^ a b c "Corpus callosum disorders". National Organization for Disorders of the Corpus Callosum. Retrieved April 11, 2010.
^ Badano JL, Mitsuma N, Beales PL, Katsanis N (2006). "The Ciliopathies: An Emerging Class of Human Genetic Disorders". Annual Review of Genomics and Human Genetics. 7: 125–148. doi:10.1146/annurev.genom.7.080505.115610. PMID 16722803.
^ Dominguez, R; Aguirre Vila-Coro, A; Slopis, JM; Bohan, TP (June 1991). "Brain and ocular abnormalities in infants with in utero exposure to cocaine and other street drugs". American Journal of Diseases of Children. 145 (6): 688–95. doi:10.1001/archpedi.1991.02160060106030. PMID 1709777.
^ Martin GN, Carlson NR, Buckist W (2007). Psychology (3rd ed.). London: Pearson Education Company.
External linksEdit
Classification
D
OMIM: 217990
MeSH: D061085
DiseasesDB: 29900
External resources
eMedicine: radio/193
Patient UK: Agenesis of the corpus callosum
Retrieved from "https://en.wikipedia.org/w/index.php?title=Agenesis_of_the_corpus_callosum&oldid=1049049023"


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The Platform may contain links to third party websites, advertisers, products, services, offers, or other events or activities that are not owned or controlled by Lenovo. Lenovo does not assume any responsibility for any such third party sites, information, materials, products, services or offers. If you access any third party website, product, service, or content from the Platform, you do so at your own risk and you agree that Lenovo will have no liability arising from your use of or access to any third party website, product, service, or content.
Trademarks and Ownership of Intellectual Property
Lenovo™, the Lenovo logo, and certain other product names and phrases are trademarks or service marks of Lenovo in the U.S. and/or other countries. The absence of a trademark or service mark designation next to a product or service name or logo belonging to Lenovo anywhere on the Platform does not constitute a waiver of Lenovo’s trademark or other intellectual property rights concerning that name or logo used or referenced on the Platform.
The Platform contains content owned or licensed by Lenovo ("Lenovo Content"). Lenovo Content is protected by copyright, trademark, trade secret and other laws, and, as between you and Lenovo, Lenovo owns and retains all copyright, trademark, trade secret, patent and other intellectual property rights in and to Lenovo Content and the Platform (the “Intellectual Property”). Lenovo reserves all rights in the Lenovo Content and Intellectual Property not expressly granted in these Terms. Lenovo does not permit the use of the Lenovo Content or its Intellectual Property in advertising, as an endorsement for any product or service, or for any other purpose, commercial or otherwise, without the prior express written permission of Lenovo.
Termination
You may terminate use of the Platform at any time for any reason (in other words, un-register). Lenovo may terminate or suspend your right to use the Platform at any time, with or without cause or notice to you. Lenovo may terminate the Platform at any time without notice. Upon termination, all licenses and other rights granted to you in these Terms will immediately cease. Following termination, Lenovo may retain your Content for a commercially reasonable period of time for backup, archival, or audit purposes. Furthermore, Lenovo and its other users may retain and continue to use, store, display, reproduce, modify, create derivative works of, perform, and distribute any of your Content that other users have stored or shared through the Platform.
Indemnification
You agree to defend, indemnify and hold harmless Lenovo and its employees, officers, affiliates and agents from and against any claims, liabilities, damages, losses, and expenses, including without limitation, reasonable attorney's fees and costs, arising out of or in any way connected with any of the following: (i) Your Content or your misuse of the Platform; (ii) your breach or alleged breach of these Terms; (iii) your violation of any third party right, including without limitation, any intellectual property right, publicity, confidentiality, property or privacy right; (iv) your violation of any laws, rules, regulations, codes, statutes, ordinances or orders of any governmental authorities, including, without limitation, all regulatory, administrative and legislative authorities; or (v) any misrepresentation made by you. You will cooperate as reasonably requested by Lenovo with respect to any such claim. Lenovo reserves the right to assume control of the defence or settlement of any such claim, and you will not in any event settle any such claim without the prior written consent of Lenovo.
Governing Law
These Terms are governed by the laws of England.
Entire Agreement; Severability
These Terms, together with Lenovo’s Data Privacy Policy and any amendments and any additional agreements you may enter into with Lenovo in connection with the Platform, shall constitute the entire agreement between you and Lenovo concerning the Platform. If any provision of these Terms is deemed invalid, then that provision will be limited or eliminated to the minimum extent necessary, and the remaining provisions of these Terms will remain in full force and effect.
Limitations of Liability
Neither Lenovo nor its affiliates, officers, directors, employees, agents, third party content providers, sponsors, or licensors warrant that the Site will be continuous, uninterrupted or error-free. THE PLATFORM, INCLUDING WITHOUT LIMITATION, ITS CONTENT, IS PROVIDED ON AN "AS IS" AND "AS AVAILABLE" BASIS. TO THE FULLEST EXTENT PERMISSIBLE BY LAW, LENOVO HEREBY DISCLAIMS ALL WARRANTIES, EXPRESS OR IMPLIED, INCLUDING, BUT NOT LIMITED TO, THE WARRANTIES OF MERCHANTABILITY, FITNESS FOR A PARTICULAR PURPOSE OR NON-INFRINGEMENT, OR ANY WARRANTIES BASED ON A COURSE OF DEALING. TO THE MAXIMUM EXTENT PERMITTED BY LAW, LENOVO SHALL NOT BE LIABLE FOR ANY INDIRECT, INCIDENTAL, SPECIAL, CONSEQUENTIAL OR PUNITIVE DAMAGES, OR ANY LOSS OF PROFITS OR REVENUES, OR ANY LOSS OF DATA OR USE, RESULTING FROM YOUR ACCESS TO OR USE OF OR INABILITY TO ACCESS OR USE LEGION COMMUNITY, EVEN IF LENOVO HAS BEEN ADVISED OF THE POSSIBILITY OF ANY SUCH DAMAGES IN ADVANCE.
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Legion Community Terms and Conditions
Last updated 19th February 2021
Welcome to Legion Community (“Legion Community” or the “Platform") - powered by Standing on Giants*- an online communication platform operated by Lenovo (the “Client”).
Legion Community enables its registered users (the “Users") to post and share content, such as information about Lenovo products, services, events, social media activity and videos ("Content”) to the Platform.
Legion Community is provided by Lenovo through the services of Standing on Giants subject to these Terms (“Terms”). We reserve the right to amend these Terms at any time and without notice. If we amend the Terms, we will post the amended Terms on this page and indicate at the top of the page the date the Terms were last revised. Your continued use of the Platform after any such changes constitutes your acceptance of the new Terms.
By accessing or using Legion Community you signify that you have read, understood, and agree to be bound by these Terms. If you do not agree, then you should not access or use the Platform.
Lenovo’s decision in relation to any aspect of Legion Community and these Terms is final and binding.
Your Use of Legion Community Platform
ELIGIBILITY. This Program is open to Participants who are age 18 years or older at the time of entry. This Program is not open to: (1) employees or internally contracted vendors of [Client] or its parent/subsidiaries, agents or affiliates; (2) the immediate family members or members of the same household of any such employee or vendor; (3) anyone professionally involved in the development or administration of this Program; (4) employees or internally contracted vendors of governments and government-affiliated companies or organizations; or (5) any employee whose employer's guidelines or regulations do not allow entry in the Program. This Program is open to residents of Italy, Spain, Norway, Finland, Sweden, Denmark, Kingdom of Saudi Arabia, United Arab Emirates, South Africa. This Program is void in any other country and where otherwise prohibited or restricted by law.
HOW TO PARTICIPATE. To participate in the Program, visit accurately and truthfully complete the online registration form, agree to the terms and conditions of this Agreement, and follow the other instructions at the website listed above.
REWARDS. Subject to the terms and conditions of this Agreement, and once confirmed by [Client], Participants will be able to accrue “points” that can be redeemed for reward(s). Rewards will be featured at various price points and in limited quantities.
The approximate retail values of rewards are subject to change based on current market conditions at the time of reward redemption. Participants are not entitled to any surplus between actual retail value (ARV) of a reward and stated ARV and any difference between stated ARV and actual value of the reward will not be awarded. No substitution, assignment, transfer, or cash redemption of any reward is allowed. Lenovo reserves the right to substitute a reward with another reward of equal or greater value should the advertised reward become unavailable for any reason. If applicable, rewards may be fulfilled in the form of voucher(s), coupon(s) and/or gift card(s) in Lenovo’s sole discretion. If a Participant is unable to participate in or accept a reward or any portion of a reward for any reason, Lenovo shall have no further obligation to such Participant. Lenovo will not replace any lost or stolen rewards after redemption by Participants. In no event will Lenovo be responsible for fulfilling more than the stated number of rewards. Participants acknowledge and agree that rewards are available in limited quantities and reward type and availability are subject to change at Lenovo’s sole discretion. Lenovo shall have no obligation or liability to Participants for any changes made to reward type or availability.
Subject to your compliance with these Terms, Lenovo grants you a non-exclusive, non-transferable, limited right to access and use the Platform only for your personal, informational, and non-commercial use. You agree not to interrupt or attempt to interrupt the operation of the Platform in any way or circumvent or attempt to circumvent any security feature of the Platform. Your use of the information and Content available on the Platform is subject to the following restrictions:
You must not modify or remove any and all copyright, trademark or other proprietary notices contained in the Content.
You must not modify the Content in any way or reproduce or publicly display, perform, or distribute or otherwise use the Content for any commercial purpose.
No title or ownership rights in the Content or any intellectual property rights therein are transferred to you under these Terms. You may never use another user's account without the permission of that user. When creating your account, you must provide accurate and complete information and you must keep your account password secure. You must notify Lenovo immediately of any breach of security or unauthorised use of your account. Lenovo will not be liable for any and all losses caused by any unauthorised use of your account
You agree not to collect or harvest any personal data from the Platform including any personally identifiable information such as but not limited to account names, nor to use the communication systems provided by the Platform for any commercial solicitation purposes. You agree not to use any portion of the Platform as a destination linked from any unsolicited bulk messages or unsolicited commercial messages.
Your Content Posted or Linked to Legion Community
Lenovo allows you to submit posts or link content to Legion Community, including photos, images, videos, sounds, comments, links, tweets, likes, and other materials. A Legion Community moderator will moderate all posts after publication. Lenovo reserves the right to remove user’s content that it considers to be irrelevant to the purpose of the platform, or that it considers violates any of these Terms. Lenovo may also post or link to the Platform any of your Content that you make available to Lenovo for the Platform. You are solely responsible for your Content that is posted or linked to the Platform and Lenovo accepts no liability for your Content.
You grant Lenovo and other users of the Platform a non-exclusive, royalty-free, transferable, sub-licensable, worldwide license to use, store, display, reproduce, modify, create derivative works of, perform, publish, re-post, re-tweet, and distribute your Content on or in connection with the Platform.
You represent and warrant that you have all rights necessary in your Content for it to be on or available from the Platform, and that the posting and use of your Content on or through the Platform does not violate, misappropriate or infringe on the rights of any third party, including, without limitation, privacy rights, publicity rights, copyrights, trademark and/or other intellectual property rights. You further represent and warrant to the extent there are links on the Platform to your Content that is hosted on third party websites, such linking is permitted by the terms of service of any such third party websites. You agree to comply with all laws, rules and regulations applicable to your use of the Platform and your Content, including but not limited to copyright laws.
You agree that your Content will not include any violent, discriminatory, unlawful, infringing, hateful, defamatory, derogatory, harassing, pornographic or obscene photos, text, images, messages, references, content or other materials. In addition, your Content will not be confidential or proprietary, and Lenovo will have no confidentiality obligation regarding your Content.
You agree that Lenovo is only acting as a passive conduit for your online distribution and publication of your Content. Lenovo does not have any obligation to review your Content, and therefore does not guarantee the accuracy, integrity, or quality of all user Content and we cannot assure you that harmful, inaccurate, deceptive, offensive, threatening, defamatory, unlawful, or otherwise objectionable Content will not appear on the Platform. Lenovo is not responsible for any public display or misuse of your Content. Lenovo does however reserve the right to remove, exclude or modify your Content for any reason, at its discretion, including any Content that Lenovo believes violates these Terms or other applicable policies. UNDER NO CIRCUMSTANCES WILL LENOVO BE LIABLE TO YOU OR ANY THIRD PARTY IN ANY WAY FOR ANY CONTENT POSTED ON OR MADE AVAILABLE THROUGH A SITE BY YOU OR ANY OTHER USER.
Lenovo may reward points for certain activities on the Platform, including (but not limited to) sharing posts, creating posts and entering competitions. Subject to availability, Lenovo reserves the right at any time to amend, modify or retract any points, rewards or prizes earned on the Platform. You accept that Lenovo will not enter into any correspondence regarding the awarding of points or rewards and Lenovo is entitled to grant or revoke rewards at their discretion. The decision of Lenovo in relation to all aspects of this programme is final and binding.
Data Privacy and Security
Lenovo will communicate with you via email or push notifications (including via the Standing on Giants platform), about Legion Community related notices changes to features of Legion Community and any notices required by law, in lieu of communication by postal mail.. If required by law, we will ask you for consent before sending you marketing emails. Please note that you may unsubscribe to general marketing emails at any time using the link provided in the emails. You understand that your data may be shared with other Lenovo entities and trusted third parties with a contractual relationship with Lenovo solely in connection with Legion Community
Please read our Data Privacy Policy for more information on our privacy and security practices.
Representations and Warranties
You represent and warrant that:
(a) You will abide by and comply with these Terms;
(b) Your Content and Lenovo’s use thereof as contemplated by these Terms and the Platform will not infringe any rights of any third party, including but not limited to any intellectual property rights, privacy rights, and rights of publicity;
(c) You have the full power and authority to enter into these Terms and to the extent that any entity is bound hereby, to bind such entity, these Terms, and performance of obligations under these Terms do not and will not violate any other agreement to which you or such entity is a party; and
(d) You will comply with all applicable laws and regulations, including but not limited to those set forth in these Terms.
Compliance
When using Legion Community, you will comply with all applicable laws and regulations, and all terms of use and similar rules concerning your use of any Social Media Outlet or other third party publishing platform.
Upon our request made to you from time to time, you will cooperate with us and allow us access to your systems, communications, and records solely in connection with determining your compliance with these Terms.
Visiting Third Party Websites
The Platform may contain links to third party websites, advertisers, products, services, offers, or other events or activities that are not owned or controlled by Lenovo. Lenovo does not assume any responsibility for any such third party sites, information, materials, products, services or offers. If you access any third party website, product, service, or content from the Platform, you do so at your own risk and you agree that Lenovo will have no liability arising from your use of or access to any third party website, product, service, or content.
Trademarks and Ownership of Intellectual Property
Lenovo™, the Lenovo logo, and certain other product names and phrases are trademarks or service marks of Lenovo in the U.S. and/or other countries. The absence of a trademark or service mark designation next to a product or service name or logo belonging to Lenovo anywhere on the Platform does not constitute a waiver of Lenovo’s trademark or other intellectual property rights concerning that name or logo used or referenced on the Platform.
The Platform contains content owned or licensed by Lenovo ("Lenovo Content"). Lenovo Content is protected by copyright, trademark, trade secret and other laws, and, as between you and Lenovo, Lenovo owns and retains all copyright, trademark, trade secret, patent and other intellectual property rights in and to Lenovo Content and the Platform (the “Intellectual Property”). Lenovo reserves all rights in the Lenovo Content and Intellectual Property not expressly granted in these Terms. Lenovo does not permit the use of the Lenovo Content or its Intellectual Property in advertising, as an endorsement for any product or service, or for any other purpose, commercial or otherwise, without the prior express written permission of Lenovo.
Termination
You may terminate use of the Platform at any time for any reason (in other words, un-register). Lenovo may terminate or suspend your right to use the Platform at any time, with or without cause or notice to you. Lenovo may terminate the Platform at any time without notice. Upon termination, all licenses and other rights granted to you in these Terms will immediately cease. Following termination, Lenovo may retain your Content for a commercially reasonable period of time for backup, archival, or audit purposes. Furthermore, Lenovo and its other users may retain and continue to use, store, display, reproduce, modify, create derivative works of, perform, and distribute any of your Content that other users have stored or shared through the Platform.
Indemnification
You agree to defend, indemnify and hold harmless Lenovo and its employees, officers, affiliates and agents from and against any claims, liabilities, damages, losses, and expenses, including without limitation, reasonable attorney's fees and costs, arising out of or in any way connected with any of the following: (i) Your Content or your misuse of the Platform; (ii) your breach or alleged breach of these Terms; (iii) your violation of any third party right, including without limitation, any intellectual property right, publicity, confidentiality, property or privacy right; (iv) your violation of any laws, rules, regulations, codes, statutes, ordinances or orders of any governmental authorities, including, without limitation, all regulatory, administrative and legislative authorities; or (v) any misrepresentation made by you. You will cooperate as reasonably requested by Lenovo with respect to any such claim. Lenovo reserves the right to assume control of the defence or settlement of any such claim, and you will not in any event settle any such claim without the prior written consent of Lenovo.
Governing Law
These Terms are governed by the laws of England.
Entire Agreement; Severability
These Terms, together with Lenovo’s Data Privacy Policy and any amendments and any additional agreements you may enter into with Lenovo in connection with the Platform, shall constitute the entire agreement between you and Lenovo concerning the Platform. If any provision of these Terms is deemed invalid, then that provision will be limited or eliminated to the minimum extent necessary, and the remaining provisions of these Terms will remain in full force and effect.
Limitations of Liability
Neither Lenovo nor its affiliates, officers, directors, employees, agents, third party content providers, sponsors, or licensors warrant that the Site will be continuous, uninterrupted or error-free. THE PLATFORM, INCLUDING WITHOUT LIMITATION, ITS CONTENT, IS PROVIDED ON AN "AS IS" AND "AS AVAILABLE" BASIS. TO THE FULLEST EXTENT PERMISSIBLE BY LAW, LENOVO HEREBY DISCLAIMS ALL WARRANTIES, EXPRESS OR IMPLIED, INCLUDING, BUT NOT LIMITED TO, THE WARRANTIES OF MERCHANTABILITY, FITNESS FOR A PARTICULAR PURPOSE OR NON-INFRINGEMENT, OR ANY WARRANTIES BASED ON A COURSE OF DEALING. TO THE MAXIMUM EXTENT PERMITTED BY LAW, LENOVO SHALL NOT BE LIABLE FOR ANY INDIRECT, INCIDENTAL, SPECIAL, CONSEQUENTIAL OR PUNITIVE DAMAGES, OR ANY LOSS OF PROFITS OR REVENUES, OR ANY LOSS OF DATA OR USE, RESULTING FROM YOUR ACCESS TO OR USE OF OR INABILITY TO ACCESS OR USE LEGION COMMUNITY, EVEN IF LENOVO HAS BEEN ADVISED OF THE POSSIBILITY OF ANY SUCH DAMAGES IN ADVANCE.
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Candidate: Dr. Robert Michael Sutton, Assistant Professor of Anesthesia, Critical Care, and Pediatrics at the University of Pennsylvania (UPenn), and Pediatric Critical Care Attending Physician at the Children's Hospital of Philadelphia (CHOP), is applying for a Mentored Patient-Oriented Research Career Development Award (K23). This candidate has an established record of academic and research excellence, and the investigation proposed in this application is a natural progression of his growing expertise in assessing CPR quality. The candidate will conduct a prospective interventional trial evaluating the existing quality of in-hospital pediatric CPR and the effectiveness of defibrillators with automated audiovisual feedback to improve compliance with American Heart Association (AHA) pediatric CPR guidelines. Subsequently, through an analytic approach novel to pediatric resuscitation research, the candidate will obtain data to challenge the existing CPR guidelines, which have been developed by expert consensus using available animal and adult data. This investigation will place the candidate at the forefront of pediatric CPR quality research, prime his career for future independent investigations, and establish a research niche that is complementary to yet distinct from his accomplished mentors. Dr. Sutton aspires to become an independent resuscitation scientist working in a leading academic institution with greater than 75% time committed to patient-oriented resuscitation research. His long term goal is to lead a research team focused specifically on improving CPR process with the overall goal of improving survival and quality of life outcomes for children who suffer a cardiopulmonary arrest. The candidate's career activities during this award have been selected to ensure his transition to independent resuscitation scientist. Environment: The career development plan and the intended research investigation build upon an existing network of research and mentorship already in place at the candidate's institution. Experts in resuscitation research have developed a clinical learning laboratory to study cardiac arrest. The candidate has played an integral role in the development of these networks and will continue to collaborate and receive mentorship from this talented group. As a student in the Center for Clinical Epidemiology and Biostatistics at UPenn, the candidate will continue training in epidemiologic methods, but will also have extensive statistical resources committed to the completion of the research investigation. In short, the environment available to the candidate coupled with an established and internationally recognized team of mentors, all but guarantee the success of the proposed career development award. Research: Context: Cardiac arrests in children are a major public health problem. The quality of healthcare provider CPR during adult resuscitations typically does not comply with American Heart Association (AHA) clinical practice guidelines. CPR monitoring defibrillators with audiovisual feedback improve adult CPR quality and clinical outcomes. This same technology can now practically be used to assess and improve CPR guideline compliance in younger children. Objective: Characterize the current landscape of CPR quality during in-hospital pediatric cardiac arrest, determine the effect of audiovisual feedback to improve compliance with the existing consensus-based CPR guidelines, and begin to challenge the paradigm that these consensus-based guidelines achieve acceptable physiologic outcomes associated with survival. Study Design: The design of the protocol is a prospective before-after interventional study design. The existing quality of CPR performed during pediatric resuscitations will be recorded using CPR monitoring defibrillators during a 24 month Control (""""""""Before"""""""") Period. The Intervention (""""""""After"""""""") Period represents a 24 month period during which the effect of audiovisual feedback to improve compliance with our existing consensus-based CPR guidelines will be determined. In a final exploratory aim, compliance with existing guidelines will be associated to arterial blood pressures to determine if compliance with our existing consensus-based pediatric CPR guidelines achieves acceptable physiologic outcome. Summary: The proposed timely and innovative investigation will change the landscape of pediatric resuscitation science from an evidence-poor to an evidence-rich platform. The informative data obtained coupled with formal statistical training in methods novel to resuscitation research, will place the candidate at the forefront of pediatric CPR quality research. By establishing a research niche distinct from his mentors, the candidate's career will be primed for future independent research investigations designed to improve quality of life outcomes for children who suffer a cardiac arrest.
Public Health Relevance
The quality of healthcare provider CPR during adult resuscitations typically does not comply with American Heart Association (AHA) clinical practice guidelines. The goal of this investigation is to characterize the current landscape of CPR quality during in-hospital pediatric cardiac arrest, determine the effect of audiovisual feedback to improve compliance with the existing consensus-based CPR guidelines, and change the foundation of pediatric CPR guideline development from consensus-based to evidence-based. The knowledge obtained from the timely and innovative investigation will forever change the landscape of pediatric resuscitation from an evidence-poor to an evidence-rich platform.
Funding Agency
Agency
National Institute of Health (NIH)
Institute
Eunice Kennedy Shriver National Institute of Child Health & Human Development (NICHD)
Type
Mentored Patient-Oriented Research Career Development Award (K23)
Project #
5K23HD062629-03
Application #
8326706
Study Section
Pediatrics Subcommittee (CHHD)
Program Officer
Maholmes, Valerie
Project Start
2010-09-20
Project End
2015-08-31
Budget Start
2012-09-01
Budget End
2013-08-31
Support Year
3
Fiscal Year
2012
Total Cost
$121,200
Indirect Cost
$8,599
Institution
Name
Children's Hospital of Philadelphia
Department
Type
DUNS #
073757627
City
Philadelphia
State
PA
Country
United States
Zip Code
19104
Related projects
NIH 2014
K23 HD Improving the Quality of In-hospital Pediatric Resuscitation
Sutton, Robert Michael / Children's Hospital of Philadelphia
NIH 2013
K23 HD Improving the Quality of In-hospital Pediatric Resuscitation
Sutton, Robert Michael / Children's Hospital of Philadelphia $120,006
NIH 2012
K23 HD Improving the Quality of In-hospital Pediatric Resuscitation
Sutton, Robert Michael / Children's Hospital of Philadelphia $121,200
NIH 2011
K23 HD Improving the Quality of In-hospital Pediatric Resuscitation
Sutton, Robert Michael / Children's Hospital of Philadelphia $121,855
NIH 2010
K23 HD Improving the Quality of In-hospital Pediatric Resuscitation
Sutton, Robert Michael / Children's Hospital of Philadelphia $121,855
Publications
Morgan, Ryan W; Kilbaugh, Todd J; Shoap, Wesley et al. (2017) A hemodynamic-directed approach to pediatric cardiopulmonary resuscitation (HD-CPR) improves survival. Resuscitation 111:41-47
Morgan, Ryan W; French, Benjamin; Kilbaugh, Todd J et al. (2016) A quantitative comparison of physiologic indicators of cardiopulmonary resuscitation quality: Diastolic blood pressure versus end-tidal carbon dioxide. Resuscitation 104:6-11
Sutton, Robert M; French, Benjamin; Meaney, Peter A et al. (2016) Physiologic monitoring of CPR quality during adult cardiac arrest: A propensity-matched cohort study. Resuscitation 106:76-82
Naim, Maryam Y; Sutton, Robert M; Friess, Stuart H et al. (2016) Blood Pressure- and Coronary Perfusion Pressure-Targeted Cardiopulmonary Resuscitation Improves 24-Hour Survival From Ventricular Fibrillation Cardiac Arrest. Crit Care Med 44:e1111-e1117
Sutton, Robert M; Case, Erin; Brown, Siobhan P et al. (2015) A quantitative analysis of out-of-hospital pediatric and adolescent resuscitation quality--A report from the ROC epistry-cardiac arrest. Resuscitation 93:150-7
Wolfe, Heather; Maltese, Matthew R; Niles, Dana E et al. (2015) Blood Pressure Directed Booster Trainings Improve Intensive Care Unit Provider Retention of Excellent Cardiopulmonary Resuscitation Skills. Pediatr Emerg Care 31:743-7
Wolfe, Heather; Zebuhr, Carleen; Topjian, Alexis A et al. (2014) Interdisciplinary ICU cardiac arrest debriefing improves survival outcomes*. Crit Care Med 42:1688-95
Sutton, Robert M; Friess, Stuart H; Maltese, Matthew R et al. (2014) Hemodynamic-directed cardiopulmonary resuscitation during in-hospital cardiac arrest. Resuscitation 85:983-6
Rodriguez, Silvana Arciniegas; Sutton, Robert M; Berg, Marc D et al. (2014) Simplified dispatcher instructions improve bystander chest compression quality during simulated pediatric resuscitation. Resuscitation 85:119-23
Sutton, Robert M; French, Benjamin; Niles, Dana E et al. (2014) 2010 American Heart Association recommended compression depths during pediatric in-hospital resuscitations are associated with survival. Resuscitation 85:1179-84